Hello,
35 days ago today that I self harm and I have no desire, no thought, which is why I write these words .. .
Many people fall into behaviors that are harmful to themselves, as smoking or drinking. But the main difference between these behaviors and someone who cuts his own arms repeatedly is that people do not smoke with the intention of harming. The damage is an unfortunate side effect, and the reason why smoking is pleasure. In contrast, those who intend to cut injury, I do not think this is so ..
There is also an important distinction between attempted suicide and self injury in the case of attempted suicide, the damage is uncertain and basically invisible on the contrary, when a person is cut, the damage is clear, predictable and often highly visible.
The most common are cuts in the arms, hands and legs, and less commonly the face, abdomen, breasts and even genitals. Some are burned or scalded, others beat their bodies, or bumped into something. Other ways in which people harm include scratching, stinging, biting, scraping and occasionally inserting sharp objects under the skin or body orifices or swallowing sharp objects or harmful substances.
There is a hypothesis that self-injury, results from a decrease of brain neurotransmitters. This view is justified by the evidence found by Wichelen and Stanley (1991). They found that while systems of dopamine and opioids are not involved in self lesion, the serotonin system it is. Drugs that fail to reach the brain more serotonin seem to have some effect on self-injurious behavior. Stanley Wichelen and hypothesized a relationship between this and the clinical similarities between obsessive-compulsive disorder (which is known to be drug can help increase serotonin levels) and self-injurious behaviors. They realized that some drugs that stabilize mood (such as Tegretol) can stabilize the behavior of getting hurt.
Coccaro (1997) has conducted studies to see if the serotonin system is involved in the conduct of self-injury, irritability is found that the behavior correlated with central serotonin function, and the type of aggressive behavior shown in response to irritation appears to be dependent on serotonin levels. If serotonin levels are normal, irritability expressed screaming, throwing things, etc. If serotonin levels are low, increases aggression and response to irritation is climbing towards self-injury, suicide or attack others.
Simeon et al. (1992) found that self-injurious behavior (automatically) was significantly negatively correlated with the number of binding sites of platelet imipramine (the self-injurers have fewer imipramine binding sites in platelets a level of serotonin activity) and note that this "may reflect central serotonergic dysfunction pre-synaptic inhibition of serotonin release ... serotonergic dysfunction may facilitate self injury." When these results are considered in the light of works such as Stoff et al. (1987) and Birmaher et al. (1990), which links reduced numbers of binding sites of platelet imipramine with impulsivity and aggression, it seems the most appropriate classification for self-injurious behavior may be a disorder of impulse control, similar to trichotillomania , kleptomania, or compulsive gambling. Herpertz (Herpertz et al, 1995; Herpertz and Favazza, 1997) has investigated how the levels of prolactin in the blood respond to doses of d-fenfluramine in subjects who self-harm and people who do not.
The prolactin response in subjects self-injury was poor, which suggests a deficit at all, and mainly, the central role presynaptic 5-HT (serotonin). " Stein et al. (1996) found a similar deficit in the prolactin response to fenfluramine challenge in subjects with compulsive personality disorder, and Coccaro et al. (1997c) found that prolactin response varied inversely with scores on the scale of "History of Life in Relation to Aggression." Not clear whether these abnormalities are caused by experiences of trauma / abuse / invalidation or whether some individuals with brain abnormalities such traumatic experiences are impossible for them to learn effective ways to handle stressful situations, and therefore feel they have little control over what happens to them and end up resorting to self-injury as a survival mechanism.
Many who self-harm (self-mutilate) fail to properly explain his behavior, but we do know is when to stop a session of self-injury. After a certain amount of wounds, the need or despair is somehow resolved, and the person feels peaceful, calm, at peace. In one study (Richardson and Zaleski, 1986) gave the self-injury naloxone, a drug that blocks the action of reducing pain of endorphins produced naturally by the body. The results indicate that there was effective ie still report feeling little or no pain. The pain affects not stop injuries. Haines et al. (1995) found that the reduction of psycho-physiological stress is the main purpose of self-injury. When a certain comfort level is achieved physiological, the self-harmed no longer feel the urge to harm your body. The lack of pain may be due to the dissociation that occurs at the time, or in that self-injury serves to focus 100% on something concrete and other stimuli are blocked, including physical and emotional pain.
Dissociation can be defined as that state of mind in which the individual has a temporary disconnection of the self-awareness, time or of external circumstances. It has been found increased theta activity in some dissociative mental states called "altered states of consciousness", such as the hypnotic trance and meditation. Moreover, it has reported an increase in the power of the theta band during the anticipation of a painful stimulus. Brain activity that originates in the middle of the prefrontal cortex and anterior cingulate structures limbic-related cognitive-affective processing of pain. When you build an experimental painful stimuli, changes in cerebral blood flow (CBF) in the anterior cingulate cortex (ACC) is related directly to the perception of unpleasantness of the stimulus. The activation of this structure is critical for emotional and behavioral reaction of pain, given its proximity to the premotor structures or of performance linked to processes of care and avoidance behavior. The integration of the three areas of pain, expressed as intensity, level of disgust and affection secondary, takes place in a central system of areas that process nociceptive information serial and parallel.
An EEG finding important in patients with BPD who have CAL is the increase in the theta band in the basal state and during the cold tolerance test. In patients with BPD who do not perceive pain during the CAL, the power of the theta band correlated directly with the total score of the dissociative experiences scale.
BPD patients reporting no pain during the CAL have lower scores on the reports of intensity and aversive component when subjected to experimental tests of acute pain. In addition, patients report improved mood states such as depression, anger, anxiety and confusion applied immediately after nociceptive testing. They also present a higher degree of impulsivity and dissociation, and also higher number of suicide attempts.
I miss you and talk therapy, the practice of listening to the feelings, remember? There is talk of listening to the soul, or female principle (which represents the emotions in man) and also to hear the messages of the unconscious. You'll tell me what you think of all this that I write ...
A hug.
PS: They say that the pilgrim starts from the moment you think of the way ...
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